Anabel Flores Arenas, Ph.D.
Assistant Professor of Biology
Office Phone: 951-343-4873 E-mail: firstname.lastname@example.org Office Location: James 382 Office Hours: T & TH 7:30 - 8:30 AM, 12:30-1:30 PM; W 10:45 - 11:45 AM; F 9:00 - 11:00 AM (virtual) or By appointment
|Ph.D.||Cell and Molecular Biology||University of Michigan||2019|
|B.S.||Biology and Spanish||California Baptist University||2010|
Post-baccalaureate Research Education Program (PREP)
Wake Forest University
BIO143 Principles of Biology with Lab
WLD121 Science Technology and Worldview
BIO489 Immunology and Molecular Pathogenesis with Lab
BIO490 Undergraduate Research
BIO590 Graduate Research
BIO153 Anatomy and Physiology I Lab
BIO163 Anatomy and Physiology II Lab
Research, Presentations, & Publications
Kumaraswamy M, Riestra A, Flores A, Uchiyama S, Dahesh S, Bondsäter G, Nilsson V, Chang M, Seo H, Sakoulas G, Nizet V. Unrecognized Potent Activities of Colistin Against Clinically Important mcr+ Enterobacteriaceae Revealed in Synergy with Host Immunity. bioRxiv [Preprint]. 2023 Mar 21:2023.03.21.533661. doi: 10.1101/2023.03.21.533661. PMID: 36993410; PMCID: PMC10055327.
Argetsinger LS, Flores A, Svezhova N, Ellis M, Reynolds C, Cote JL, Cline JM, Myers MG, Carter-Su C. Role of the Beta and Gamma Isoforms of the Adapter Protein SH2B1 in Regulating Energy Balance. Endocrinology. 2023 Mar 13;164(5):bqad032. doi: 10.1210/endocr/bqad032. PMID: 36799031.
Hassan S, Pullikuth A, Nelson KC, Flores A, Karpova Y, Baiz D, Zhu S, Sui G, Huang Y, Choi YA, Sadelain M, D.Agostino R, Hemal A, Holzen U, Debinski W, and Kulik G. “Beta2-adrenoreceptor signaling increases therapy-resistance in prostate cancer by upregulating MCL1.” Mol Cancer Res. (2020).
Cote JL, Argetsinger LS, Flores A, DeSantis L, Bedard A, Myers MG Jr, Carter-Su C. “Mice lacking the brain-specific alpha and delta isoforms of scaffold protein SH2B1 have reduced body weight and are resistant to a high fat diet.” Diabetes. (2020).
Flores, A, Argetsinger, LS, Stadler LKJ, Malaga, A, Vander, PB, DeSantis, LC, Joe RM, Cline JM, Keoth JM, Henning E, Barroso I, Mendes de Oliveira E, Chandrashekar G, Clutter, ES, Hu Y, Stuckey J, Farooqi IS, Myers, MG Jr, and Carter-Su, C. “Crucial Role of the SH2B1 PH Domain for the Control of Energy Balance.” Diabetes. 68:11 (2019) (Cover and Spotlight article).
Joe R., Flores A, Cline JM, Clutter ES, Vander P., Doche ME, Argetsinger LS, Shoelson S, and Carter-Su C. “The C-terminal tail of SH2B1α inhibits the ability of SH2B1 to enhance NGF functions and alters SH2B1 subcellular localization, both by a phosphorylation-dependent mechanism.” Mol. Cell Biol. 38: e00277-17 (2017) (Cover and Spotlight article).
Hassan S, Karpova Y, Flores A, D’Agostino R Jr, Danhauer SC, Hemal A, and Kulik G. “A pilot study of blood epinephrine levels and CREB phosphorylation in men undergoing prostate biopsies.” Int Urol Nephrol. 46(3):505-10 (2014).
Hassan S, Karpova Y, Flores A, D'Agostino R Jr, and Kulik G. “Surgical stress delays prostate involution in mice.” PLoS One. 6;8(11): e78175 (2013).
Baiz D, Hassan S, Choi YA, Flores A, Karpova Y, Yancey D, Pullikuth A, Sui G, Sadelain M, Debinski W, and Kulik G. “Combination of the PI3K inhibitor ZSTK474 with a PSMA-targeted immunotoxin accelerates apoptosis and regression of prostate cancer.” Neoplasia.15(10):1172-83 (2013).
Yancey D, Nelson KC, Baiz D, Hassan S, Flores A, Pullikuth A, Karpova Y, Axanova L, Moore V, Sui G, and Kulik G. “BAD dephosphorylation and decreased expression of MCL-1 induce rapid apoptosis in prostate cancer cells.” PLoS One.8(9): e74561(2013.
Hassan S, Karpova Y, Baiz D, Yancey D, Pullikuth A, Flores A, Register T, Clark M, D’Agostino R, Danial N, Datta SR, and Kulik G. “Stress inhibits apoptosis and accelerates prostate cancer development in mice through BAD phosphorylation.” J Clin Invest. 123(2):874-86 (2013).
Community Service & Involvement
Red Cross Club Advisor
Education First co-founder
Interests & Hobbies
Running, hiking, traveling, classical music, and trying new foods.
What, then, shall we say in response to these things? If God is for us, who can be against us?